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Other aspects of treatment vary according to the cause of liver failure (Table 3)

Notably, this neuroprotective profile was abolished at supraphysiological concentrations (5001000 nM), where paradoxical potentiation of L-VSCC currents coincided with loss of cytoprotective effects

Thinking one could substitute for the other in a research context would be a catastrophic error in study design

In this study, 141 treatment-nave patients with chronic HBV infection, 38 patients who achieved HBsAg loss following antiviral treatment, and 47 patients who suffered from HBV-related HCC from real-life clinical practice were recruited