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Uptake of both apoptotic and eryptotic cells via recognizing externalized PS molecules suggests that the effects on macrophage reprogramming might be similar
These results are similar to those reported by Atlas (2021), and they demonstrate that NAC-amide supplementation, in addition to decreasing the aggregation of A peptides, manages to attenuate the carbonylation of A proteins and also inhibits the phosphorylation of MAPK, JNK and p38MAPK pathways, leading to reversal of A1-42-induced neurotoxicity, suggesting a neuroprotective effect of NAC [45,49,69]
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