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These data indicate that sUA may trigger the NLRP3 inflammasome in different but complementary manners, such as inducing mitochondrial ROS production, potassium efflux and through changes in cell membrane morphology

Animal-model studies published in peer-reviewed literature report that localized IGF-1 analog administration measurably accelerates healing in models of muscle laceration, contusion, and strain injury

Researchers continue investigating how NNMT-related mechanisms may affect obesity, glucose metabolism, and energy expenditure

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