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Treatment of these tolerogenic DCs improves experimental autoimmune myasthenia (EAMG) via repressing expansion of lymphocyte cells, development of Treg cells, transformation of T helper1 and T helper17 to T helper2 cells and reducing pathogenic antibody levels [67]

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Additionally, as noted by Mates et al., weight loss, loss of muscle or fat, and protein malnutrition are key indicators that may suggest the presence of thiamine deficiency

Strategies to address this include designing biocompatible dendrimers, adjusting surface charges, and applying chemical modifications to mitigate toxicity for clinical applications [201]