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Research suggests GHK-Cu helps transition macrophages from the pro-inflammatory M1 phenotype toward the anti-inflammatory, pro-healing M2 phenotype at appropriate stages of repair

(PubMed) Andorfer JH, Tchaikovskaya T, Listowsky I

Importantly, a major target for SFN-mediated protein oxidation was STAT3, a transcription factor involved in the regulation of T H 17-related genes

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