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Second, elevated lactate directly inhibits immune cell activation/proliferation and drives Kla-mediated immunosuppression, promoting CTLA-4 expression in T cells, M2-like macrophage polarization, and dendritic cell dysfunction (47)

(2018) showed that the pathophysiology of hepatic IR injury is primarily marked by lipid metabolic reprogramming, which results in a secondary effect of inflammation, thereby highlighting the role of lipid metabolism in disease pathogenesis

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