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The protective effects of liraglutide against A-induced neurotoxicity in neuroblastoma cells are attributed to the activation of the phosphoinositide 3-kinase (PI3K)/Akt signaling pathway, as evidenced by the increased phosphorylation of Akt and the altered expression of the apoptosis-related proteins Bcl-2/Bax, and cytochrome c [49]
However, its considered a triple threat because it is not solely a GLP-1
Ammonia diffuses freely across the bloodbrain barrier and is converted with alanine to glutamine by glutamine synthase
This typically represents increased activity in healing tissues rather than worsening of the condition
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