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doi:10.2147/DDDT.S358989 Fisman EZ, Tenenbaum A

It has been suggested that patients with this disease may also exhibit refusal to eat, potentially due to the accumulation of malonyl CoA in the hypothalamus, where high levels of malonyl CoA can suppress appetite and feeding [26, 27]

While the former aids in transporting fatty acids to be burned for energy within mitochondria, the latter produces acetylcholine that can cross the blood-brain barrier