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Dysfunction of BMSCs reduces their osteogenic differentiation and proliferative capacity, thereby compromising osteoblast formation and contributing to age-related declines in bone formation ( Method As shown in Figure 2 , a systematic review of recent research for ferroptosis and osteoporosis was conducted using the PubMed and Embase search engines from 1 January 1, 2019 to February 20, 2025

Therefore, the inhibition of xCT-mediated cystine transport, cystine depletion, limited cysteine biosynthesis, impaired GSH synthesis, or inactivation of GPX4 can induce ferroptosis [49]

It functions as a coenzyme in several oxidation reduction reactions ( Figure 12.6 )

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