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In summary, PLB significantly improved the survival, mitochondrial function and antioxidant capacity of KGN cells, as well as suppressed cell damage and ferroptosis by inhibiting YTHDF1 expression

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When comparing unstimulated and hydrogen peroxide-exposed chondrocytes, our protein interaction network revealed altered levels of actin-binding proteins and those regulating interactions between actin filaments and the plasma membrane, as well as between cells and the ECM