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Further, given the findings in other BPC 157 studies [1,3,4], it can be that on a molecular level, BPC 157 can interfere with increased tumor necrosis factor-alpha (TNF-alpha) and malondialdehyde (MDA) levels [8,11], suggesting ongoing increased inflammatory and oxidative processes that can be induced by dopaminergic enhancers, such as amphetamine [171]

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Defective NCOA4-dependent ferroptosis in senescent fibroblasts retards diabetic wound healing