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Silicon-induced lung injury may result from multiple interacting pathological mechanisms, including the damage caused to macrophages by the phagocytosis of SiO 2 , the reactive oxygen species (ROS) production, mitophagy and apoptosis ( Hydrogen (H 2 ), as a gaseous therapeutic agent, can directly penetrate the lungs without requiring systemic absorption, thereby exerting its effects locally

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Indeed, the structural and functional abnormalities of lysosomes are critical for the pathogenesis and development of various diseases, including tumors 14,15,16 , neurodegenerative diseases 17,18,19 , inflammatory diseases 20,21,22 , and cardiovascular diseases 23,24,25 , making LQC a novel but pivotal target for treatments

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