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In various pathological conditions implicating oxidative stress, ER functions are disturbed resulting in ER stress and initiation and propagation of unfolded protein response (UPR), which results in transient suppression of protein translation to allow recovery but leads to cell death when stress cannot be resolved (Soliman et al., 2019)

Nevertheless, the ability to selectively target GSH metabolic components in therapeutic applications remains a challenge

Oral dihexa is not pharmacologically viable because the peptide is degraded by gastric acid and pancreatic proteases before absorption, resulting in bioavailability below 5%

For ongoing benefits, weekly sessions are often recommended