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As ACSL4 gains traction as a therapeutic target in oncology, its role in activating the ferroptosis mechanism presents a novel strategy for treating tumors, especially those resistant to conventional chemotherapy or adept at evading apoptosis [43]

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Both formulations work through the same GLP-1 receptor mechanism, so both can produce similar heart rate effectsthough individual responses depend partly on genetics and dose titration speed

Stiles LI, Ferrao K, Mehta KJ