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(79) showed that promoting CB proliferation and SCFA production repairs the colonic mucosal barrier in DSS-induced mice by upregulating tight-junction proteins and increasing goblet cell numbers, dependent on CB -mediated amino acid and 2-oxocarboxylic acid metabolism

Contrasting with these actions of GIPR activation to reduce food intake, Gipr knockout (KO) mice, and humans with impaired function GIPR variants, are protected against obesity, also probably through a central effect, as similar observations were made in global KO mice or mice lacking Gipr in nestin, synapsin or VGAT-expressing cells 4,5,9,10,11,12

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Overall, Epithalon represents a promising tool in the pursuit of a healthier, longer lifespan, playing an important role in modern peptide therapy